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What are the signs symptoms classification and treatment of Adrenal Hyper Plasia

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It includes the vomiting which occurs due to the wasting of salt which may lead to the dehydration and death. There is an infertility which occurs due to the anovulation. One also observes the hyper tension. There is an excessive facial hair, virilization and irregularity in the menses mainly in adolescence period. There is an early growth of pubic hair mainly in the child hood.

What are the associated conditions of adrenal hyper plasia?

It includes the vomiting which occurs due to the wasting of salt which may lead to the dehydration and death. There is an infertility which occurs due to the anovulation. One also observes the hyper tension. There is an excessive facial hair, virilization and irregularity in the menses mainly in adolescence period. There is an early growth of pubic hair mainly in the child hood. There is condition known as the ambiguous genitalia in which there is a difficult to determine a sex and mainly occur in the females. In few cases there is an absence of puberty.

What is the classification of adrenal hyper plasia?

It includes the cortisol which is an adrenal steroid hormone and occurs due to the need for function of endocrine. It starts producing during the second month of fetal life and its poor production is a good sign of congenital adrenal hyper plasia and is referred as the CAH. A decrease in the production of cortisol may lead to the increase levels of ACTH which induce hyper plasia. There is also an over activity of the steroid producing cells of the adrenal cortex. The defects causing this disorder are mainly congenital and are present at birth. The deficiency in cortisol in congenital adrenal hyper plasia and is referred as the CAH is not complete and is a serious problem. The synthesis of cortisol occurs along with the synthesis of mineralo corticoids. It includes the aldosterone, androgens like testosterone and estrogen like estradiol. The increase or decrease in the production of three hormones which is the main causative agent in congenital adrenal hyper plasia and is referred as the CAH. There is a specific deficiency in the enzyme which shows an increase or decrease in the production of mineralo corticoids or sex hormones.

  1. In every form the congenital adrenal hyper plasia and is referred as the CAH shows a deficiency in the enzyme 21 hydroxylase. More than the 90 percent of the cases occur due to the congenital adrenal hyper plasia and is referred as the CAH. It is mainly referred to the deficiency in the enzyme 21 hydroxylase. In the first week of life there is a severe 21 hydroxylase deficiency which leads to the wasting of salt which may lead to the dehydration and death. There is a moderate 21 hydroxylase deficiency which leads to the excessive facial hair, virilization and irregularity in the menses mainly in adolescence period. There is a mild 21 hydroxylase deficiency which leads to an infertility which occurs due to the anovulation. One also observes the hyper tension. There is an early growth of pubic hair mainly in the child hood.
  2. The congenital adrenal hyper plasia and is referred as the CAH may occur due to the deficiency of other enzymes. It shows an increase or decrease in the production of mineralo corticoids or sex hormones.
  3. From the year 1960, onwards there are professionals which have given the traditional names to congenital adrenal hyper plasia and are referred as the CAH. It responds to the deficiency of enzyme. In the year 1980, the enzymes were identified by the help of genes. Most of them were cytochrome P 450 oxidase and were renamed by the way to reflect it. In few cases, more than one enzyme helps to participate in a reaction and in some cases a single enzyme mediated in many reactions. One can also see a variation in the different tissues and species.

What is the treatment of adrenal hyper plasia?

It includes the increase in the gluco corticoid so that one is able to treat the condition of hyper plasia and leads to increase in the production of mineralo corticoids or androgens. One must provide a replacement for the mineralo corticoids and an extra salt required for a person. One must also provide a replacement for the testosterone or estrogen at the time of puberty. There are other treatments required to control the growth by extending the puberty or the process of maturation of bone. There are surgeries which are linked to the genital reconstruction and can rectify the problems which occur and are related to the abnormal genital structure. All the treatment options are properly discussed in relation with the enzyme deficiency.

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